Docsity
Docsity

Prepare for your exams
Prepare for your exams

Study with the several resources on Docsity


Earn points to download
Earn points to download

Earn points by helping other students or get them with a premium plan


Guidelines and tips
Guidelines and tips

NURS 629 EXAM 4 QUESTIONS AND ANSWERS, Exams of Gynecology

NURS 629 EXAM 4 QUESTIONS AND ANSWERS 1. NURS 629 Exam 4 study guide with answers 2. How to prepare for NURS 629 Exam 4 3. NURS 629 Exam 4 practice questions and solutions 4. Common topics covered in NURS 629 Exam 4 5. NURS 629 Exam 4 review materials online 6. Tips for passing NURS 629 Exam 4 7. NURS 629 Exam 4 sample questions with explanations 8. Best resources for NURS 629 Exam 4 preparation 9. NURS 629 Exam 4 key concepts and definitions 10. How to ace NURS 629 Exam 4 with minimal stress 11. NURS 629 Exam 4 question formats and types 12. NURS 629 Exam 4 study group near me 13. Downloadable NURS 629 Exam 4 flashcards 14. NURS 629 Exam 4 previous year questions 15. Expert advice for NURS 629 Exam 4 success 16. NURS 629 Exam 4 time management strategies 17. NURS 629 Exam 4 difficulty level and pass rate 18. NURS 629 Exam 4 grading criteria explained 19. NURS 629 Exam 4 last-minute revision tips 20. Common mistakes to avoid in NURS 629 Exam 4 21. NURS 629 Exam 4 mock test

Typology: Exams

2024/2025

Available from 07/02/2025

Prof.Steve
Prof.Steve 🇺🇸

361 documents

1 / 32

Toggle sidebar

This page cannot be seen from the preview

Don't miss anything!

bg1
1 / 16
which
NURS 629 EXAM 4 QUESTIONS AND
ANSWERS
1. What are the two types of biliary atresia?
ANS fetal- noted in womb (other defects like heart, spleed, intestines)
perinatal- appears 2-4 weeks after birth
2. What is hypospadias?
ANS Hypospadias is a birth defect in boys in opening of the urethra
is not located at the tip of the penis.
3. What are the Clinical signs of premature adrenarche
ANS Premature adrenarche (PA) refers to the presentation of androgenic signs -
appearance of pubic and axillary hair, adult-type body odor, oily hair, acne or come
dones - before the age of 8 years in girls or 9 years in boys in the absence of central
puberty
4. What is physiological jaundice?
ANS -occurs when baby accumulates bilirubin
-secondary to immature liver in newborns
-common first 2-4 days of life and resolves by 2 weeks
5. What does a positive coomb's test in a pregnant mother mean
ANS may lead to physiological jaundice in child
she developed antibodies IgG and may lead to hemolytic disease in newborn
6. What level is conjugated hyperbilirubinemia?
pf3
pf4
pf5
pf8
pf9
pfa
pfd
pfe
pff
pf12
pf13
pf14
pf15
pf16
pf17
pf18
pf19
pf1a
pf1b
pf1c
pf1d
pf1e
pf1f
pf20

Partial preview of the text

Download NURS 629 EXAM 4 QUESTIONS AND ANSWERS and more Exams Gynecology in PDF only on Docsity!

which

NURS 629 EXAM 4 QUESTIONS AND

ANSWERS

1. What are the two types of biliary atresia?

ANS fetal- noted in womb (other defects like heart, spleed, intestines) perinatal- appears 2-4 weeks after birth

2. What is hypospadias?

ANS Hypospadias is a birth defect in boys in opening of the urethra is not located at the tip of the penis.

3. What are the Clinical signs of premature adrenarche

ANS Premature adrenarche (PA) refers to the presentation of androgenic signs - appearance of pubic and axillary hair, adult-type body odor, oily hair, acne or come dones - before the age of 8 years in girls or 9 years in boys in the absence of central puberty

4. What is physiological jaundice?

ANS -occurs when baby accumulates bilirubin -secondary to immature liver in newborns -common first 2-4 days of life and resolves by 2 weeks

5. What does a positive coomb's test in a pregnant mother mean

ANS may lead to physiological jaundice in child she developed antibodies IgG and may lead to hemolytic disease in newborn

6. What level is conjugated hyperbilirubinemia?

ce

ANS serum conjugated bilirubin concentration greater than 1 mg/dL if the total bilirubin is less than 5.0 mg/dL or more than 20% of the total bilirubin if the the total bilirubin is greater than 5 mg/dL (p. 862 AAP book)

7. What is breastfeeding jaundice?

ANS -due to poor intake that causes lack of stools and urine output -common in the first week and resolves once milk comes in and the infant is feeding well-more stools and urinary output -peaks around 2-3 weeks

8. How do you diagnose jaundice?

ANS -dx with a bili level of 5 mg/dL -12 mg/dL threshold for all newborns having jaundiced appearan -direct/indirect bili levels -CBC -reticulocyte count

9. How do you treat jaundice?

ANS increased intake indirect sunlight phototherapy IV fluids

10. What are other causes of jaundice?

ANS abnormal blood cell shapes (like sickle cell)

-LFTs -abdominal x-ray -abdominal US -liver bx

15. How do you treat biliary atresia?

ANS surgery (Kasai procedure) liver transplant

16. What are signs and symptoms of Intussusception?

ANS paroxysmal, episodic abdominal pain with vomiting every 5 to 30 minutes, drawing up of legs with periods of rest, bloody diarrhea

  • May have fever -Sausage like mass felt in RLQ (Dance Sign) -Ab distention and tenderness

17. What are risk factors for dehydration?

ANS GI virus vomiting/diarrhea

18. What are s/sx of dehydration?

ANS sunken anterior fontanel tachycardia and decrease cap refill decrease urine output is sensitive but nonspecific increase in urine specific gravity decrease BP- late finding=more than 10% fluid loss

19. How do you treat dehydration?

ANS if minimal, mild, moderate- oral rehydration if severe (drowsy, cold extremities, lethargic, sunken/dry eyes, very depressed an- terior fontanel, no tears, dry mouth/tongue, very decreased skin turgor, rapid/some- times impalpable pulse, decreased/unrecordable pulse, deep/rapid respiratory rate, markedly reduced urine output) - IV fluids

20. What is emesis?

ANS vomiting=symptom must distinguish from regurgitation in infants integrated response to noxious stimuli-coordinated by CNS

21. What is acute emesis?

ANS short-term abrupt onset

22. What is recurrent emesis?

ANS at least 3 episodes over 3 months chronic, relatively mild that occurs frequently

23. What is cyclic emesis?

ANS recurrent, intense episodes separated by asympto- matic periods

bloody stools abdominal cramping thirst decreased urination dizziness fatigue

28. What is chronic diarrhea?

ANS 1 or more liquid to semi-solid stools passed per day for 14+days

29. What are causes of chronic diarrhea?

ANS latrogenic- excessive fluids; <25% of calories from fat infants- formula protein intolerance toddlers- chronic, nonspecific; usually resolves by 5 years of age children/adolescents- acquired lactose intolerance viral or bacterial

30. Diagnostics for diarrhea

ANS Acute- stool cultures, CMP, CBC (if indicated by usually resolves on own) Chronic- stool cultures, CBC w/diff, CMP, ESR, lactose tolerance test

31. What is GERD?

ANS neuromuscular problem where lower esophagus and and GE sphincter (cardiac sphincter) are lax and allow for easy reguritation of gastric contents

32. What are the three classifications of GERD?

ANS 1. physiologic- infre- quent/episodic vomiting

2. functional- painless, effortless

3. pathologic- frequent with alteration in physical functioning (up to 70% of infants

<1700g have this type with 85% being symptoms free by 1 yr)

33. What are s/sx of GERD?

ANS recurrent regurgitation w/wo vomiting heartburn gas/abdominal pain failure to thrive ruminative behavior hematemesis dysphagia respiratory disorders (wheezing, stridor, cough) hoarseness feeding difficulties typically asymptomatic by 6 weeks of age

wt loss, dehydration, constipation

39. Diagnostic testing for pyloric stenosis

ANS US, upper GI series which can show a "string sign" which is a fine elongated pyloric canal "olive mass" palpable in epigastric area to right of midline Treatment is surgery (pyloromyotomy)

40. What is appendicitis?

ANS inflammation/infection of the vermiform appendix- small appendage arising from the cecum most common 6-19 yo

41. What causes appendicitis?

ANS obstruction of the appendiceal lumen (inside of appendix) usually by fecalith (fecal stone) or lymphoid hyperplasia (#of lympho- cytes) -may also be caused by inspissated secretion of CF -may also be caused by parasitic tumor or foreign body

42. Symptoms of appendicitis

ANS initial- dull, steady periumbilical pain for 4-6 hrs shifts to RLQ as peritoneal inflammation develops

24-36 hrs- rupture likely- may have initial decrease in pain with worsening shortly after *N/V pain awakens from sleep decreased appetite likely no fever before perforation

43. Diagnostic testing for appendicitis

ANS CBC w/ diff- look for shift to left, *elevat- ed WBCs usually 10-20, neutrophils, elevated bands UA CT w/ contrast- highest accuracy may use US/MRI **surgery referral, emergent

44. What is Markle's sign?

ANS Appendicitis abdominal assessment- heel drop jar- ring test, on toes for 15 sec, dropping down forcefully on heels will elicit RLQ pain

45. What is rebound tenderness?

ANS Appendicitis abdominal assessment- involun- tary guarding over McBurney's point on abdominal exam

46. What is Rosving's sign?

ANS Appendicitis abdominal assessment- pressure deep in the LLQ with sudden release elicits pain in the RLQ which strongly suggests peritoneal irritation

47. What is Psoas sign?

51. What are uncomplicated UTI symptoms?

ANS common symptoms- dysuria, urgency, frequency, no fever

52. What are complicated UTI symptoms?

ANS fever, toxicity, dehydration, child <6 months, structural abnormality

53. Diagnostic testing for UTI

ANS UA w/ culture *E.Coli most common cause dipstick= leukocytes, nitrates, RBCs if pyelo suspected- CBC, ESR, BUN, creatinine

54. Treatment for UTI

ANS trimethoprim/sulfamethoxazole (bactrim) 8-10 mg/kg/day (>2 months) *first drug of choice cefixime >= 6 yo tx 7-10 days f/u to make sure treatment effective

55. What is Osgood-Schlatter disease?

ANS caused by microtrauma in the deep fibers of the patellar tendon at insertion point -most common in later adolescence -most common with sports participation

56. How do you diagnose Osgood-Schlatter disease?

ANS physical exam, history, x-ray

57. Clinical findings for Osgood-Sclatter disease

ANS pain, swelling, tenderness at tibial tuberosity knee pain that worsens with activity tightness of the surrounding muscles, especially quads pain varies from mild with activity to nearly constant and debilitating

58. How do you treat Osgood-Schlatter

ANS decrease with quad loading and bend- ing RICE treatment quad and hamstring stretching*** NSAIDs

59. What is Legg-Calve-Perthes disease?

ANS idiopathic osteonecrosis of the femoral head onset usually 4-8 yo unilateral in 90% of cases 4x more common in males

60. Symptoms of Legg-Calve-Perthes Disease

ANS hip, thigh, or knee pain painless limp

P- pattern changes from prior headaches

66. Diagnostics for headaches

ANS headache diary CT only if tumor suspected

67. Treatment for headaches

ANS NSAIDs prophylactic CCBs, tricyclic antidepressants abortive ANS triptans not approved in children nonpharmacologic- reduce triggers

68. What are seizures?

ANS paroxysmal events thought to represent abnormal elec- trical activity in cerebral neurons usually idiopathic

69. Tonic clonic seizure

ANS tonic-entire body becomes rigid, first phase clonic- uncontrolled jerking may cry, groan, fall risk, bite inside of tongue/cheek, incontinent

70. Tonic seizure

ANS body becomes tense, usually lasts less than 20 seconds usually occurs with patient is sleeping and involve most or all of the brain

71. Absent seizures

ANS very quick, <10 seconds- often missed generalized onset involving both sides of the brain at the same time most common type stops all activity then patient stares off eyes may roll up eyelids flutter

72. Atypical absent seizures

ANS starts with patient staring off, change muscle tone and movement blinking repeatedly smacking lips or chewing movements rubbing fingers together or making other hand motions lasts longer than absent >20 seconds

73. Myoclonic seizures

ANS usually involve the neck, shoulders, upper arms usually occurs after waking up- beginning around puberty

74. Atonic seizures

ANS muscles suddenly become limp eyelids may droop head may drop forward fall risk typically lasts <15 seconds

75. Infantile seizures

ANS from birth secondary- birth trauma

78. What are observed s/sx of concussion?

ANS amnesic to event appears dazed/stunned confusion moves clumsily answers slowly loses consciousness shows mood, behavior, personality changes

79. Diagnostics for concussion

ANS based on symptoms all children with moderate head trauma (GCS 9-12) and severe (GCS 3-8) needs CT

80. Signs of normal cognition development in toddlers

ANS emerging empathy understanding social rules constructing narratives reciprocity in play

81. What are symptoms of ADHD?

ANS attention deficit hyperactivity disorder inattention hyperactivity impulsivity

82. How do you diagnose ADHD?

ANS have to have at least 6 hyperactivity/impulse symptoms and 6 inattention symptoms DSM IV

83. What is treatment for ADHD?

ANS stimulants- methylphenidate, ampheta- mine/dextroamphetamine

84. What are symptoms of sensory processing disorder in adolescents?

ANS -

overly sensitive to touch, noise, smell, or other people poor self-esteem afraid of failing at new tasks lethargic and slow always on the go impulsive distractible clumsy, slow, poor motor skills or handwriting can't find this in the book or in PP from brenda lee's study guide

85. What are comorbidities of depression?

ANS anxiety disorder can correlate with depression ADHD conduct, learning, and oppositional defiant disorders SAD