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NURS 629 EXAM 4 QUESTIONS AND ANSWERS 1. NURS 629 Exam 4 study guide with answers 2. How to prepare for NURS 629 Exam 4 3. NURS 629 Exam 4 practice questions and solutions 4. Common topics covered in NURS 629 Exam 4 5. NURS 629 Exam 4 review materials online 6. Tips for passing NURS 629 Exam 4 7. NURS 629 Exam 4 sample questions with explanations 8. Best resources for NURS 629 Exam 4 preparation 9. NURS 629 Exam 4 key concepts and definitions 10. How to ace NURS 629 Exam 4 with minimal stress 11. NURS 629 Exam 4 question formats and types 12. NURS 629 Exam 4 study group near me 13. Downloadable NURS 629 Exam 4 flashcards 14. NURS 629 Exam 4 previous year questions 15. Expert advice for NURS 629 Exam 4 success 16. NURS 629 Exam 4 time management strategies 17. NURS 629 Exam 4 difficulty level and pass rate 18. NURS 629 Exam 4 grading criteria explained 19. NURS 629 Exam 4 last-minute revision tips 20. Common mistakes to avoid in NURS 629 Exam 4 21. NURS 629 Exam 4 mock test
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ANS fetal- noted in womb (other defects like heart, spleed, intestines) perinatal- appears 2-4 weeks after birth
ANS Hypospadias is a birth defect in boys in opening of the urethra is not located at the tip of the penis.
ANS Premature adrenarche (PA) refers to the presentation of androgenic signs - appearance of pubic and axillary hair, adult-type body odor, oily hair, acne or come dones - before the age of 8 years in girls or 9 years in boys in the absence of central puberty
ANS -occurs when baby accumulates bilirubin -secondary to immature liver in newborns -common first 2-4 days of life and resolves by 2 weeks
ANS may lead to physiological jaundice in child she developed antibodies IgG and may lead to hemolytic disease in newborn
ANS serum conjugated bilirubin concentration greater than 1 mg/dL if the total bilirubin is less than 5.0 mg/dL or more than 20% of the total bilirubin if the the total bilirubin is greater than 5 mg/dL (p. 862 AAP book)
ANS -due to poor intake that causes lack of stools and urine output -common in the first week and resolves once milk comes in and the infant is feeding well-more stools and urinary output -peaks around 2-3 weeks
ANS -dx with a bili level of 5 mg/dL -12 mg/dL threshold for all newborns having jaundiced appearan -direct/indirect bili levels -CBC -reticulocyte count
ANS increased intake indirect sunlight phototherapy IV fluids
ANS abnormal blood cell shapes (like sickle cell)
-LFTs -abdominal x-ray -abdominal US -liver bx
ANS surgery (Kasai procedure) liver transplant
ANS paroxysmal, episodic abdominal pain with vomiting every 5 to 30 minutes, drawing up of legs with periods of rest, bloody diarrhea
ANS GI virus vomiting/diarrhea
ANS sunken anterior fontanel tachycardia and decrease cap refill decrease urine output is sensitive but nonspecific increase in urine specific gravity decrease BP- late finding=more than 10% fluid loss
ANS if minimal, mild, moderate- oral rehydration if severe (drowsy, cold extremities, lethargic, sunken/dry eyes, very depressed an- terior fontanel, no tears, dry mouth/tongue, very decreased skin turgor, rapid/some- times impalpable pulse, decreased/unrecordable pulse, deep/rapid respiratory rate, markedly reduced urine output) - IV fluids
ANS vomiting=symptom must distinguish from regurgitation in infants integrated response to noxious stimuli-coordinated by CNS
ANS short-term abrupt onset
ANS at least 3 episodes over 3 months chronic, relatively mild that occurs frequently
ANS recurrent, intense episodes separated by asympto- matic periods
bloody stools abdominal cramping thirst decreased urination dizziness fatigue
ANS 1 or more liquid to semi-solid stools passed per day for 14+days
ANS latrogenic- excessive fluids; <25% of calories from fat infants- formula protein intolerance toddlers- chronic, nonspecific; usually resolves by 5 years of age children/adolescents- acquired lactose intolerance viral or bacterial
ANS Acute- stool cultures, CMP, CBC (if indicated by usually resolves on own) Chronic- stool cultures, CBC w/diff, CMP, ESR, lactose tolerance test
ANS neuromuscular problem where lower esophagus and and GE sphincter (cardiac sphincter) are lax and allow for easy reguritation of gastric contents
ANS 1. physiologic- infre- quent/episodic vomiting
<1700g have this type with 85% being symptoms free by 1 yr)
ANS recurrent regurgitation w/wo vomiting heartburn gas/abdominal pain failure to thrive ruminative behavior hematemesis dysphagia respiratory disorders (wheezing, stridor, cough) hoarseness feeding difficulties typically asymptomatic by 6 weeks of age
wt loss, dehydration, constipation
ANS US, upper GI series which can show a "string sign" which is a fine elongated pyloric canal "olive mass" palpable in epigastric area to right of midline Treatment is surgery (pyloromyotomy)
ANS inflammation/infection of the vermiform appendix- small appendage arising from the cecum most common 6-19 yo
ANS obstruction of the appendiceal lumen (inside of appendix) usually by fecalith (fecal stone) or lymphoid hyperplasia (#of lympho- cytes) -may also be caused by inspissated secretion of CF -may also be caused by parasitic tumor or foreign body
ANS initial- dull, steady periumbilical pain for 4-6 hrs shifts to RLQ as peritoneal inflammation develops
24-36 hrs- rupture likely- may have initial decrease in pain with worsening shortly after *N/V pain awakens from sleep decreased appetite likely no fever before perforation
ANS CBC w/ diff- look for shift to left, *elevat- ed WBCs usually 10-20, neutrophils, elevated bands UA CT w/ contrast- highest accuracy may use US/MRI **surgery referral, emergent
ANS Appendicitis abdominal assessment- heel drop jar- ring test, on toes for 15 sec, dropping down forcefully on heels will elicit RLQ pain
ANS Appendicitis abdominal assessment- involun- tary guarding over McBurney's point on abdominal exam
ANS Appendicitis abdominal assessment- pressure deep in the LLQ with sudden release elicits pain in the RLQ which strongly suggests peritoneal irritation
ANS common symptoms- dysuria, urgency, frequency, no fever
ANS fever, toxicity, dehydration, child <6 months, structural abnormality
ANS UA w/ culture *E.Coli most common cause dipstick= leukocytes, nitrates, RBCs if pyelo suspected- CBC, ESR, BUN, creatinine
ANS trimethoprim/sulfamethoxazole (bactrim) 8-10 mg/kg/day (>2 months) *first drug of choice cefixime >= 6 yo tx 7-10 days f/u to make sure treatment effective
ANS caused by microtrauma in the deep fibers of the patellar tendon at insertion point -most common in later adolescence -most common with sports participation
ANS physical exam, history, x-ray
ANS pain, swelling, tenderness at tibial tuberosity knee pain that worsens with activity tightness of the surrounding muscles, especially quads pain varies from mild with activity to nearly constant and debilitating
ANS decrease with quad loading and bend- ing RICE treatment quad and hamstring stretching*** NSAIDs
ANS idiopathic osteonecrosis of the femoral head onset usually 4-8 yo unilateral in 90% of cases 4x more common in males
ANS hip, thigh, or knee pain painless limp
P- pattern changes from prior headaches
ANS headache diary CT only if tumor suspected
ANS NSAIDs prophylactic CCBs, tricyclic antidepressants abortive ANS triptans not approved in children nonpharmacologic- reduce triggers
ANS paroxysmal events thought to represent abnormal elec- trical activity in cerebral neurons usually idiopathic
ANS tonic-entire body becomes rigid, first phase clonic- uncontrolled jerking may cry, groan, fall risk, bite inside of tongue/cheek, incontinent
ANS body becomes tense, usually lasts less than 20 seconds usually occurs with patient is sleeping and involve most or all of the brain
ANS very quick, <10 seconds- often missed generalized onset involving both sides of the brain at the same time most common type stops all activity then patient stares off eyes may roll up eyelids flutter
ANS starts with patient staring off, change muscle tone and movement blinking repeatedly smacking lips or chewing movements rubbing fingers together or making other hand motions lasts longer than absent >20 seconds
ANS usually involve the neck, shoulders, upper arms usually occurs after waking up- beginning around puberty
ANS muscles suddenly become limp eyelids may droop head may drop forward fall risk typically lasts <15 seconds
ANS from birth secondary- birth trauma
ANS amnesic to event appears dazed/stunned confusion moves clumsily answers slowly loses consciousness shows mood, behavior, personality changes
ANS based on symptoms all children with moderate head trauma (GCS 9-12) and severe (GCS 3-8) needs CT
ANS emerging empathy understanding social rules constructing narratives reciprocity in play
ANS attention deficit hyperactivity disorder inattention hyperactivity impulsivity
ANS have to have at least 6 hyperactivity/impulse symptoms and 6 inattention symptoms DSM IV
ANS stimulants- methylphenidate, ampheta- mine/dextroamphetamine
overly sensitive to touch, noise, smell, or other people poor self-esteem afraid of failing at new tasks lethargic and slow always on the go impulsive distractible clumsy, slow, poor motor skills or handwriting can't find this in the book or in PP from brenda lee's study guide
ANS anxiety disorder can correlate with depression ADHD conduct, learning, and oppositional defiant disorders SAD