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Neurological Disorders Exam: Key Concepts & Clinical Applications, Exams of Neurology

A comprehensive overview of neurological disorders, covering key concepts, clinical manifestations, diagnostic procedures, and treatment approaches. It includes a series of questions and answers that test understanding of various neurological conditions, such as stroke, seizures, multiple sclerosis, parkinson's disease, myasthenia gravis, amyotrophic lateral sclerosis, huntington's disease, trigeminal neuralgia, and bell's palsy. Valuable for students and professionals seeking to deepen their knowledge of neurological disorders and their management.

Typology: Exams

2024/2025

Available from 02/04/2025

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tizian-kylan 🇺🇸

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NURS 3525- Neurological Disorders Exam With Complete
Solutions 2025-2026
Types
of
stroke
ischemic
and
hemorrhagic
non
modifiable
risk
factors
for
strokes
age,
gender,
ethnicity,
race
(African
American),
family
hx
modifiable
risk
factors
for
strokes
HTN,
heart
dz
(a-fib),
smoking,
DM,
sleep
apnea,
lack
of
exercise,
poor
diet,
drug/alcohol
use
Transient
Ischemic
Attack
(TIA)
minit
strokes,
blood
supply
to
part
of
the
brain
is
blocked
for
a
period
and
then
a
full
recovery
T/F
TIAs
don't
increase
risk
for
stokes
false,
they
do
increase
the
risk
for
stokes
types
of
ischemic
stroke
thrombotic
(clot
formation)
and
embolic
(clot
that
has
traveled)
clinical
manifestations
for
strokes
decreased
motor
function,
communication,
affect,
intellectual
function,
spatial
pf3
pf4
pf5
pf8
pf9
pfa

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NURS 3525- Neurological Disorders Exam With Complete

Solutions 2025- 2026

Types of stroke ischemic and hemorrhagic non modifiable risk factors for strokes age, gender, ethnicity, race (African American), family hx modifiable risk factors for strokes HTN, heart dz (a-fib), smoking, DM, sleep apnea, lack of exercise, poor diet, drug/alcohol use Transient Ischemic Attack (TIA) minit strokes, blood supply to part of the brain is blocked for a period and then a full recovery T/F TIAs don't increase risk for stokes false, they do increase the risk for stokes types of ischemic stroke thrombotic (clot formation) and embolic (clot that has traveled) clinical manifestations for strokes decreased motor function, communication, affect, intellectual function, spatial

perceptual problems, elimination issues receptive aphasia can't understand what is being said expresive aphasia slurred speech or inability to speak global aphasia When both production and understanding of language is damaged T/F stoke patient are emotional true elimination issues with a stroke constipation, and bladder control Diagnostic tests fro strokes non-contrast head CT or MRI CTA or MRA cardiac imaging what is the use of CTA and MRA in stroke diagnostics? looks at vessels and shows exactly where the stroke is taking place TX for ischemic stroke tPA (<3-4.5 hours from onset of symptoms)

secondary HA HA w/a cause tension HA Feels like a band is abound head migraine Usually unilateral and associated w/ N/V cluster HA Pain is in and around one eye use of HA diary Triggers, aura, onset, S/S, frequency triggers for HA Light, caffeine, smoking, hormones, fluctuations medications for HA Aspirin, Tylenol, NSAIDs, sumatriptan (Imitrex) prophylactic Tx for HA Anti seizure meds and botox when to call the MD for a HA? if the HA is different than normal, not responsive to medication, or accompanied by a fever complication is meningitis generalized seizures

tonic-clonic grand mal absence petit mal four phases of a seizure prodromal, aural, ictal, postictal origin of generalized seizure generalized what is the most common seizure? tonic-clonic tonic clonic seizure tonic- the muscles stiffen clonic-jerking of the muscles absence seizure muscle jerking is not present, gazing off, or non-purposeful movement focal seizures focal seizures with awareness simple partial focal seizure with impaired awareness complex partial origin of foal seizures localized complications of seizures status epileptics, depression, physical injury impaired awareness seizures awake, but not aware of self. Repetitive non-purposeful movement, may strike at physical contact

S/S of MS vision changes, weakness, care meat sign lumbar pain-a MS hug medications for MS immunosuppressants, immune modulators, monoclonal antibodies, IV steroids Use of immune modulators prevent T-cell activation and crossing the blood brain barrier characteristics of Parkinson disease progressive, neurodestructive, unknown cause, Dopamine < acetylcholine T.R.A.P for parkinsons S/S T- temors (Shakes R- rigidity (stiffness of the limbs, neck or trunk) A- akinesia (loss or impairment in power of voluntary movement) P- posture and balance issues non-motor S/S of Parkinson disease depression, fatigue, hypophonia, sleep issues, memory changes complications of parkinson disease dysphagia, problems with mobility, orthostatic HoTN, weakness, depression hallucinations, dementia

medications for Parkinson disease dopamine (carbidopa/levadopa). nticholinergics. surgical options for parkinson disease deep brain stimulation, ablations goal of Parkinson's care maintain health, encourage independence, avoid complications Myathenia gravis (MG) autoimmune, thymus involvement, fluctuating weakness of skeletal muscles S/S of MG muscle weakness, eyelid dropping, double vision, impaired speech, difficulty swallowing, changes in facial expressions, trouble breathing diagnostics for MG H&P, endrophonium tets, CT scans management of MG medications, thymectomy, plasmapheresis, IV IGB, education medications for MG anticholinergics, corticosteroids, immunosuppressants education for MG medication adherence, balanced diet, complication, adverse reactions complications of MG aspiration, pneumonia, falls, respiratory destress

does Huntington disease have excess ACE or dopamine? dopamine type of treatment for Huntington disease palliative care trigeminal neurologia unilateral severe brief stabbing recurrent pain within distribution of the CN V cause of triennial neuralgia artery compressing nerve tx for trigeminal neuralgia anti seizure and tricyclic antidepressants what to assess with trigeminal neuralgia nutritional and oral hygiene status trigeminal neuralgia triggers touch, movement, cold air, eating/chewing, brushing teeth bells palsy sudden, usually temporary paresis due to impairment of facial nerve CN VII diagnostics for Bell's palsy HX and ruling out those disorders, MRI/CT

nursing care for Bell's palsy alleviation of symptoms, prevention of complications, protection of eye on effected side