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NURS 3366 EXAM 3 REVIEW/NURS 3366 EXAM 3 REVIEW/NURS 3366 EXAM 3 REVIEW/NURS 3366 EXAM 3 REVIEW/NURS 3366 EXAM 3 REVIEW/NURS 3366 EXAM 3 REVIEW
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EXAM 3 REVIEW: Cardiovascular, Pulmonary, Renal, Genitourinary Right atrium tricuspid valve right ventriclepulmonic valvepulmonary artery pulmonary arteriolespulmonary capillaries [gas exchange with alveoli occurs here] pulmonary venules pulmonary veinsleft atriummitral valveleft ventricle aortic valve aorta part of this freshly oxygenated blood goes into coronary arteries, which branch off the aorta just beyond the aortic valve. part goes up to brain via carotids rest goes to tissue beds of remainder of body arterioles capillaries [gas exchange with tissue occurs here] venules veinsinferior vena cava (from body) or superior vena cava (from head) right atrium ❖ PERIPHERAL VASCULAR SYSTEM ➢ Venous : vessels that carry deoxygenated blood from tissues to right side of heart ▪ Healthy veins: Facilitate good venous return & prevent backflow; ▪ Bad Veins: bad valves ⟶ backflow ⟶ venous congestion ⟶ peripheral tissue edema ; stasis & immobility ⟶ sluggish blood flow ▪ Chronic Venous Insufficiency (CVI): backflow (congestion) of blood into distal veins of legs & feet
mentation, cardiac output)
overdrive of RAAS
◆ SNS overdrive: epinephrine elevated ⟶ stimulates beta receptors of heart ⟶ tachycardia and/or increased contractility of heart ⟶ greater cardiac output & ejection pressure ⟶ greater driving pressure ⟶ increase in BP ◆ RAAS overdrive: low BP or volume ⟶ RAAS always “in gear” ⟶ sustained vasoconstriction & larger blood volume (retention of NA+ and H2O) ⟶ high BP ➢ Sequela: chronic high BP ⟶ hypertrophy & hyperplasia of arterial muscles ⟶ smaller lumen ⟶ more HTN ⟶ intima (inner lining) damage ⟶ inflammatory response (mediators) ⟶ increase swelling & permeability of intima endothelial cells
➢ Atrial fibrillation (A-fib): chaotic impulses in atria cause quivering (irregular heart beat); usually begins b/c myocardium has to endure long-term hypoxic strain or chronic HF ; common in elderly ▪ Sequela:
▪ S&S: angina (ischemic pain; chest pain, left arm, jaw, back pain) w/ exercise, cardiac ischemia (SOB, nausea, diaphoresis, dysrhythmias); can lead to MI if not reversed
fails)
- Dx: ANP & BNP secreted by RA & LV to lower blood vol but kidneys don’t listen ⟶ high BNP levels
◆ LV decreased contractility/pumping, high afterload/SVR, high preload/vol ⟶ decreased CO/perfusion ⟶ RAAS triggered ⟶ peripheral arterial vasoconstriction ⟶ even more afterload ◆ Pulmonary edema CAUSED BY LFH & retrograde/backup flow (First HF ⟶ Then lung probs)
▪ Vomiting ⟶ loss of HCl/accum of alkali ⟶ alkalosis ⟶ Hypoventilation ⟶ Lungs hang on to CO ➢ Inhalation & Restrictive Lung Diseases ▪ Inhalation norm: diaphragm drops ⟶ muscles pull outward ⟶ thorax enlarges ⟶ negative pressure ⟶ air sucked into bronchi & alveoli ⟶ O2 passes across membranes into blood & CO2 passes across membrane from blood into alveoli
puncture to lung ⟶ air into thorax ⟶ positive pressure ⟶ collapses lung ⟶ chest pain, SOB ➢ Closed/tension pneumothorax: trauma from inside; i.e. rupture of dz alveoli ⟶ positive pressure ⟶ collapses lung ⟶ puts pressure on other lung ⟶ chest pain, SOB ➢ Tx: chest tube ⟶ water seal ⟶ air escapes thorax and can’t get back in ⟶ negative pressure re-established ⟶ lung re-expands
▪ Asthma: chronic inflammatory disorder of airways due to bronchial hyperresponsiveness to stimuli (allergens)
spread to anus) ➢ Patho: HSV penetrates local nerve fibers ⟶ travels to spinal ganglion & lies dormant ⟶ goes back down to genitals during times of stress ⟶ breakouts of lesions ➢ Note: once infection there is no cure; stays permanent in body & breaks out periodically ➢ S&S of HSV2: painful, red, crusty crops of lesions; systemic S&S fever and malaise ➢ Tx: antivirals to treat S&S but no cure