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GASTRITIS' PATHOPHYSIOLOGY, DISEASE MANAGEMENT and DIAGNOSTIC MEASURES
Typology: Schemes and Mind Maps
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Acute gastritis: alcohol, histamine, digitalis use metabolic disorders H.pylori inflammation increased or decreased gastric secretions. Chronic: loss of t-cell gastric mucosa degredation decrease of parietal cells and intrinsic factor.
Infection with the bacterium Helicobacter pylori. inflammatory drugs (NSAIDs) such as aspirin and ibuprofen.Erosive gastritis may be caused by use of nonsteroidal anti- 20 percent of people who chronically use NSAIDs develop gastric issues. Drinking alcohol, using cocaine, exposure to radiation (erosive gastritis). Stresses such as trauma, major surgery, severe burns, or a critical illness may cause erosive/stress gastritis
GI endoscopy histology of sample tissue biopsy obtained in endoscopy Serological tests for H. pylori and breath test
Prednisolone antacids histamine 2 (H2) blockers proton pump inhibitors (PPIs) D/C NSAIDS H. pylori, antibiotics may be prescribed for up to fourteen days. PPIs are also used to treat stress gastritis.
Epidemiology: H. pylori discovered in childhood, hygiene, household density, cooking habits lead to risks associated for the disorder. 5% autoimmune in origin, 50% of cases tend to be childhood to adolescence.