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A comprehensive overview of various liver and brain disorders, including stroke, traumatic brain injury, increased intracranial pressure, brain tumors, hepatitis, cirrhosis, liver damage, liver transplant, hemochromatosis, and portal hypertension. It covers key aspects such as assessment, interventions, treatments, common medications, and interdisciplinary care planning. Particularly useful for students in healthcare fields, providing a detailed guide to understanding and managing these conditions.
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o Liver enzymes can be elevated with conditions such as diabetes, obesity, autoimmune disorders, some viral infections (especially hepatitis), and some genetic diseases. o Prothrombin and hemoglobin prior to liver biopsy
segments. Casts also immobilize the muscles and joints next to the fracture site, and may cause neurovascular compromise, malunion, skin breakdown, and compartment syndrome
f. Take blood pressure g. Assess level of consciousness and pupillary response, weakness or paralysis of extremities Treatment for anthrax RED: Priority I or emergent care is needed for victims who need immediate treatment, such as those with cardiac or respiratory distress, trauma and bleeding, or neurological deficits YELLOW: Priority II or urgent care is needed for victims who need treatment within 2 hours, such as clients with simple fractures, lacerations, or fevers; these victims should be reevaluated every 30 to 60 minutes GREEN: Priority III or non-urgent care is needed for victims who need treatment that can wait for hours; those with sprains, rashes, and minor pain should be reevaluated every 1-2 hrs. ORANGE tag indicates a client who has a non-emergent psychiatric condition BLACK: Victims who are deceased should be labeled with a black tag and transported to designated temporary morgue
Refractory - - Death is inevitable. Failure of compensating mechanisms. No response to treatment. Risk of cardiac arrest. A rising serum lactate is an indicator of inadequate tissue perfusion due to metabolic acidosis. Low arterial oxygen content, chest pain, cardiac dysrhythmias, altered LOC, ↓urinary output. MAPB cannot be maintained without assistance. ASSESSMENT AND CARE OF SHOCK SYNDROME Clinical findings correlated with organs compromised by inadequate oxygen. Regardless of the type of shock, it leads to a SBP of less than 90 mm Hg and the narrowing of pulse pressure that is inadequate to meet the tissue needs. (SBP may be elevated initially.) Myocardial infarction – Coronary artery blood flow is blocked by atherosclerotic narrowing, thrombus formation, or (less frequently) persistent vasospasm; myocardium supplied by arteries is deprived of O2; persistent ischemia may rapidly lead to tissue death" Main cause is coronary artery disease (CAD), buildup of atherosclerotic plaque in coronary arteries that restricts blood flow to heart a. Nonmodifiable risk factors include age, gender, family history, and ethnic background b. Modifiable risk factors include smoking, obesity, stress, elevated cholesterol, diabetes mellitus, and hypertension ECG (12-lead): ST elevation, accompanied by T-wave inversion in leads that monitor affected area of heart; these changes resolve as treatment progresses Lab findings : elevated troponins (early or late diagnosis); elevated CK-MB isoenzymes over 5% (early diagnosis); or elevated LDH with “flipped” isoenzymes (late diagnosis)" Signs and Symptoms: N/V or just nausea, Diaphoresis and dizziness, chest pain lasting more than 15min (squeezing or pressure) and may extend to shoulder, left arm, back or jaw, anxiety and feelings of doom Women: fatigue, sleep disturbance, shortness of breath, back pain, upper abdominal pain, epigastric pain, and nausea with or without vomiting are the most commonly identified atypical symptoms seen in women suffering from an AMI. INTERVENTIONS :
Angina pain quality is typically described as pressure, heavy, squeezing, constrictive, suffocating, vise-like, or “like an elephant sitting on my chest.” Angina is rarely described as pain. Episodes may vary from mild to severe. Several symptoms may accompany angina pain including nausea, diaphoresis, SOB, fatigue, dizziness, weakness, and anxiety. Anginal pain is not stabbing or sharp and does not change with respirations, position change, or pressure applied to the chest wall. To demonstrate anginal pain, the patient may place a clenched fist over the sternum. This is referred to as the Levine’s sign, the universal sign for angina.