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a. Blood does not flow through the heart at this time.
b. The atria are contracting
c. The valves are closed
d. It occurs during ventricular systole Ans>> The atria are contracting
a. Ejection fraction
b. Compliance
c. Preload
d. Afterload Ans>> Afterload
a. Stimulates secretion of Na+ (and thus decreases urine volume) from the kidneys
b. Stimulated the release of Na+ (thus increases urine volume) from the kidneys
c. Increases vascular smooth muscle contraction (vasoconstriction)
d. Stimulates increased heart rate, contractility, and cardiac output Ans>> Stimulate the release of Na+ (thus increasing urine volume) from the kidneys
A. Deep vein thrombosis
B. Spinal shock
C. Superior vena cava syndrome
D. Carcinoid syndrome Ans>> Superior Vena Cava Syndrome
A. External Hematoma
B. Atherosclerosis with hypertension
C. Genetics
D. Congestive heart failure Ans>> Atherosclerosis with hypertension
A. Fibrous plaque
B. Complicated plaque
C. Fatty streak
D. Aneurysm Ans>> Complicated plaque
A. The onset of atrial systole to the completion of atrial diastole
B. The onset of atrial systole to the onset of ventricular diastole
C. The onset of atrial systole to the onset of ventricular diastole
D. The onset of atrial systole to the onset of ventricular systole Ans>> The onset of atrial systole to the onset of ventricular systole
A. An advancing failure of systemic circulation as the heart weakens
B. Rapidly progressive hypertension with a diastolic pressure of 140 mmHg or more
C. A pulse pressure of greater than 60
D. Blood pressure readings consistently greater than 140/90 mmHg Ans>> Rapidly progressive hypertension with a diastolic pressure of 140 mmHg or more
A. Caused by coronary artery disease
B. A sign of peripheral artery disease
C. An attack of vasospasm brought on by the cold
D. A result of peripheral neuropathy Ans>> A sign of peripheral artery disease
a. A result of over activation of the intrinsic and extrinsic clotting system systemically
b. Characterized by attacks of vasospasm in the small arteries and arterioles of the fingers and toes
c. Strongly associated with smoking
d. Associated with janeway lesions and Oslers Nodes Ans>> Strongly associated with smoking
a. Congestive heart failure
b. Collagen vascular diseases such as scleroderma
c. Pulmonary hypertension
d. Myxedema or smoking
d. Prostacyclins, which are diminished by NSAIDs, are vasoconstrictors. Ans>> Prostacyclins, which are diminished by NSAIDS, are vasoconstrictors
A. Does not affect the cardiac index
B. Is equal to ESV - EDV
C. Does not vary much
D. None of the above are true Ans>> None of the above are true
A. Ventricular depolarization, AV
B. Ventricular depolarization, SL
C. Ventricular repolarization, AV
D. Ventricular repolarization, SL
E. None of the above Ans>> Ventricular repolarization, SL
A. 10-20%
B. 40-50%
C. 70-75%
D. 100% Ans>> 70-75%
A. Ca++
B. ATP
C. K+
D. Epinephrine
A. Obesity
B. Venous Stasis
C. Venous Endothelial damage
D. Hypercoagulable states Ans>> Obesity
A. Rhythmicity
B. Quiescence
C. Refractory period
D. Afterload Ans>> Refractory period
a. Calcium channel blockers
b. Beta blockers
c. Ace inhibitors or ARBs
d. Diuretics such as Thiazide Ans>> Beta Blockers
a. Inverted T waves and Q waves
b. Peaked T waves
c. Marked ST segment elevation
d. Absence of troponin I or T Ans>> Marked ST segment elevation
A. Transformed fibroblasts
B. Cytokine secreting adipose cells
C. Macrophages that engulf oxidized LDL
D. Endothelial cells that produce the gas nitric oxide Ans>> Macrophages that engulf oxidized LDL
A. During atrial systole
B. When the AV valves are closed
C. When the SL valves are open
D. During diastole Ans>> When the AV valves are closed
A. SA node to AV node to bundle branches to othe Bundle of HIS to purkinje fibers
B. SA node to AV node to Purkinje fibers to bundle of HIS to bundle branches
C. SA node to bundle of HIS to AV node to bundle branches to purkinje fibers
D. SA node to AV node to bundle of HIS to bundle branches to purkinje fibers Ans>> SA node to AV node to bundle of HIS to bundle branches to purkinje fibers
A. Inferior vena cava
B. Superior vena cava
C. Pulmonary artery
D. Pulmonary Vein
E. Inferior vena cava and superior vena cava
A. An increase in cardiac contractility
B. An increase in speed of heart rate and electrical conduction through the heart
C. An increase in preload
D. Both an increase in contractility and heart rate / conduction speed Ans>> Both an increase in contractility and heart rate / conduction speed
A. Non-STEMI (Non-ST elevation myocardial infarction)
B. STEMI (ST segment elevation myocardial infarction)
C. Transmural myocardial infarction
D. Myocardial stunning Ans>> Non-STEMI (Non-ST elevation myocardial infarction)
a. Ventricular depolarization
b. Ventricular repolarization
c. Atrial depolarization
d. Atrial repolarization Ans>> Ventricular repolarization
a. Mean arterial pressure (MAP)
b. Heart rate
c. Sympathetic activity
d. Afterload