Docsity
Docsity

Prepare for your exams
Prepare for your exams

Study with the several resources on Docsity


Earn points to download
Earn points to download

Earn points by helping other students or get them with a premium plan


Guidelines and tips
Guidelines and tips

Bell's palsy and stroke can both cause facial weakness or paralysis, making them difficult to distinguish initially. However, there are significant differences between the two conditions:   Bell's Palsy Cause: Temporary facial nerve paralysis, often due to inflammation or viral infection. Symptoms: Facial drooping or weakness on one side Difficulty closing the eye on the affected side   Loss of taste on the affected side of the tongue   Increased sensitivity to sound   Stroke Cause: Disruption of blood flow to the brain, often due to a blood clot or bleeding.   Symptoms: Facial drooping or weakness on one side   Slurred speech or difficulty understanding speech   Weakness or numbness in one arm or leg, especially on one side   Sudden severe headache Vision problems, such as double vision or blurred vision

Typology: Cheat Sheet

2022/2023

Uploaded on 09/17/2024

abieshaabiola
abieshaabiola 🇺🇸

4 documents

1 / 1

Toggle sidebar

This page cannot be seen from the preview

Don't miss anything!

bg1
Abiesha Smith Neuroanatomy Wednesday, March 1st, 2023
HOMEWORK FIVE
BELL’S PALSY AND STROKE
QUESTION:
1. EXPLAIN THE DIFFERENCES BETWEEN BELL’S PALSY AND A STROKE
Both Bell’s Palsy and a Stroke are medical conditions often accompanied by
facial paralysis of varying degrees and regions. But whilst the two are non-
mutually exclusive, they are not identical and cannot be substituted for the
other. Bell’s Palsy, also know as Idiopathic Peripheral Facial Paralysis, is a
deterioration of ipsilateral facial muscles that results from a disruption in
function/injury of the facial nerve after said nerve exits the cerebral area
known as the pons. In Bell’s Palsy, not only could the facial nerve become
injured just after leaving the pons but could damage could also occur in
branches of the facial nerves, resulting in a slew of varying physiological
ramifications. If the muscles of the forehead (upper face) and the lower face
—all of which are innervated by the facial nerve—are affected, full paralysis
or weakness of that particular (ipsilateral) side of the facial muscles being
innervated would occur (the characteristic droopy facial expression for which
Bell’s Palsy is known): additional complications include smoothness in the
forehead, retinal, and nasal regions of the face. A Stroke, on the other hand,
is a disruption in function/deterioration of contralateral facial muscles that
results from a lesion in the facial motor nucleus and damage to upper motor
neurons. In a Stroke, when a lesion of upper motor neurons—originating from
the right side of the cerebral cortex—occurs, the lower left side of the face is
affected due to the contralateral synapsing of said upper motor neurons to
the lower motor neurons responsible for innervation of facial muscles on the
lower left side of the face. What separates a Stroke from Bell’s Palsy is a lack
of physiological damage to the upper face in a Stroke: the forehead region,
in a Stroke, is unaffected if the ipsilateral innervation of its muscles (from the
ipsilateral cerebral cortex) is also unaffected by the lesion. As such, where
ipsilateral damage to the upper and lower faces are characteristic of Bell’s
Palsy—due to damage of any region of the facial nerve, its canals, and
innervated lower motor neuron regions, thereof—a Stroke’s paralysis, be it
upper or lower facial regions and left/right side of the face, will depend on
the upper motor neurons being affected and whether said disruptions are
either contralateral, ipsilateral, or both.

Partial preview of the text

Download Bell's Palsy & Stroke and more Cheat Sheet Neuroscience in PDF only on Docsity!

Abiesha Smith Neuroanatomy Wednesday, March 1st, 2023

HOMEWORK FIVE

BELL’S PALSY AND STROKE

QUESTION:

1. EXPLAIN THE DIFFERENCES BETWEEN BELL’S PALSY AND A STROKE

Both Bell’s Palsy and a Stroke are medical conditions often accompanied by facial paralysis of varying degrees and regions. But whilst the two are non- mutually exclusive, they are not identical and cannot be substituted for the other. Bell’s Palsy, also know as Idiopathic Peripheral Facial Paralysis , is a deterioration of ipsilateral facial muscles that results from a disruption in function/injury of the facial nerve after said nerve exits the cerebral area known as the pons. In Bell’s Palsy, not only could the facial nerve become injured just after leaving the pons but could damage could also occur in branches of the facial nerves, resulting in a slew of varying physiological ramifications. If the muscles of the forehead (upper face) and the lower face —all of which are innervated by the facial nerve—are affected, full paralysis or weakness of that particular (ipsilateral) side of the facial muscles being innervated would occur (the characteristic droopy facial expression for which Bell’s Palsy is known): additional complications include smoothness in the forehead, retinal, and nasal regions of the face. A Stroke, on the other hand, is a disruption in function/deterioration of contralateral facial muscles that results from a lesion in the facial motor nucleus and damage to upper motor neurons. In a Stroke, when a lesion of upper motor neurons—originating from the right side of the cerebral cortex—occurs, the lower left side of the face is affected due to the contralateral synapsing of said upper motor neurons to the lower motor neurons responsible for innervation of facial muscles on the lower left side of the face. What separates a Stroke from Bell’s Palsy is a lack of physiological damage to the upper face in a Stroke: the forehead region, in a Stroke, is unaffected if the ipsilateral innervation of its muscles (from the ipsilateral cerebral cortex) is also unaffected by the lesion. As such, where ipsilateral damage to the upper and lower faces are characteristic of Bell’s Palsy—due to damage of any region of the facial nerve, its canals, and innervated lower motor neuron regions, thereof—a Stroke’s paralysis, be it upper or lower facial regions and left/right side of the face, will depend on the upper motor neurons being affected and whether said disruptions are either contralateral, ipsilateral, or both.