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What is the most reliable method of confirming and montioring correct placement of an ET tube? - ANSWER-Continuous waveform capnography The upper airway consists of... - ANSWER-Nose, Mouth, Jaw, Oral Cavity, Pharynx, and Larynx No gas exchange occurs here __________, it's called ____________. - ANSWER-Nose to terminal bronchioles, anatomical dead space. (2ml/kg of inspired tidal volume) They conduct airflow towards gas exchange units. Crycothyroid membrane - ANSWER-between thyroid and cricoid, avascular structure that connects the thyroid and cricoid cartilage. Site of CRiCOTHYROTOMY- an emergency opening of the airway. A PaCO2 greater than 45 mmHg indicates: A. Metabolic acidosis. B. Metabolic alkalosis. C. Respiratory acidosis. D. Respiratory alkalosis. - ANSWER-C. Respiratory acidosis PaCO2 normal range - ANSWER-35-45 mm Hg Less than 35 likely means hyperventilation Tracheal deviation AWAY from the affected side, decreased breath
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What is the most reliable method of confirming and montioring correct placement of an ET tube? - ANSWER-Continuous waveform capnography The upper airway consists of... - ANSWER-Nose, Mouth, Jaw, Oral Cavity, Pharynx, and Larynx No gas exchange occurs here __________, it's called ____________. - ANSWER-Nose to terminal bronchioles, anatomical dead space. (2ml/kg of inspired tidal volume) They conduct airflow towards gas exchange units. Crycothyroid membrane - ANSWER-between thyroid and cricoid, avascular structure that connects the thyroid and cricoid cartilage. Site of CRiCOTHYROTOMY- an emergency opening of the airway. A PaCO2 greater than 45 mmHg indicates: A. Metabolic acidosis. B. Metabolic alkalosis. C. Respiratory acidosis. D. Respiratory alkalosis. - ANSWER-C. Respiratory acidosis PaCO2 normal range - ANSWER- 35 - 45 mm Hg Less than 35 likely means hyperventilation Tracheal deviation AWAY from the affected side, decreased breath sounds, and hyperresonance... What's happening? - ANSWER-Tension pneumothorax In a tension pneumothorax tracheal deviation goes in what direction? - ANSWER- AWAY from affected side. Normal mean pulmonary artery pressure - ANSWER- 10 - 20 mmHg Pulmonary hypertension is a mean PA pressure greater than... - ANSWER-(PAm) greater than 20 Primary pulmonary hypertension - ANSWER-Idiopathic genetic disorder caused by abnormal structure of the pulmonary blood vessels Name three causes of secondary pulmonary hypertension.. - ANSWER-1. Passive PH- the result of back pressure. Mitral Stenosis, LV systolic failure.
STEMI diagnosis - ANSWER-Chest pain + positive cardiac enzyme (TROP. >0.4), and -
Diastolic BP of at least 25mmhg in infants and at least 35 mmhm in children correlates with better outcomes. PALS Brady with a pulse - ANSWER-Assess airway, breathing, mental status Most common cause is hypoxia! could also be hypothermia and or medications. s/s of shock? AMS? hypotensive? Start CPR if any of these Always start CPR if HR < 60 bpm iv access Give Epi 0.01 mg/kg (0.1ml of 0.1mg/ml solution) Repeat Q 3-5 minutes Initial management of pediatric respiratory distress or Failure A - ANSWER-1. A-ABC. Support open airway: Comfort or Head tilt chin lift. Jaw thrust. Clear airway if indicated. (suction nose or mouth if indicated) Consider OPA or NPA. IDENTIFY type and Severity of respiratory problems Initial management of pediatric respiratory distress or Failure B - ANSWER-2. B-Monitor Spo2 withPulse ox. Provide high concentration O2, via non rebreather
PALS Management of acute asthma Mild to Moderate - ANSWER--Administer humidified O2 in high concentration via nasal cannnula or O2 mask. K
Ventilation Management - ANSWER-a Tidal volume is 5-7mL/Kg aprox. 500ml for an adult
hemodynamic changes in Cardiogenic shock - ANSWER-SBP (Down) SVR (UP) CVP (UP) CO (Down) PAP (UP) Wedge (UP) PVR (UP) Coags (PT/INR/PTT) - ANSWER-PT 11-14s PTT (20-40 sec) heparin INR (0.9-1.2) Coumadin Platelets : 150-450k Blood Gas - ANSWER-pH: 7.35-7. PaCO2: 35- 45 PaO2: 80- 100 HCO3: 22- 26 chemistry panels (renal, hepatic, comprehensive, metabolic) - ANSWER-Na+ 135- 145 Cl- 95 - 105 K+ 3.5-4. Cr 0.6-1. Glucose 70- 100 Magnesium 1.7-2. Magnesium - ANSWER-1.7-2. K+ (potassium) - ANSWER-3.5-5.0 mEq/L Na+ - ANSWER- 135 - 145 mEq/L Glucose - ANSWER- 70 - 110 mg/dL Cr (Creatinine) - ANSWER-0.6-1.3 mg/dL Systemic Vascular Resistance (SVR) - ANSWER-the force opposing the movement of blood within the blood vessels [(MAP-CVP) / CO] x Normal: 750-1600 dynes/sec SVR Formula & Normal - ANSWER-(MAP - CVP / ) x 80
Spontaneous Intermittent Mandatory Ventilation SIMV - ANSWER--Has a preset rate /minute ventilation Allows patient to over breathe a set Spontaneous breaths are not supported, so tidal volume varies based on what the patient can pull Pressure Control Ventilation - ANSWER-Set pressure - Machine is set to deliver a certain pressure over a certain I-time. pressure remains constant Tidal Volume changes as lungs change Pressure Support Ventilation (PSV) - ANSWER--Used to lower the work of spontaneous breathing and augment a patients spontaneous tidal volume
Left bundle branch... - ANSWER-Is what actually depolarizes the intraventricular septum In lead II, depolarization of the septum ( LBB) is what part of the ekg? - ANSWER-The negative deflection known as the Q wave! The Q wave is indicative of intraventricular Septal depolarization The R the wave is indicative of - ANSWER-Ventricular depolarization SaO2 - ANSWER- 95 - 100% percent of hemoglobin that is saturated with oxygen. Adult Acls Dopamine infusion dose - ANSWER- 2 - 10 mcg/ kg / min Adult ACLS Epi infusion dose - ANSWER- 2 - 10 mcg/kg/min Adult ACLS Bradycardia Atropine dose - ANSWER-1st dose: 0.5 mg bolus Repeat q 3-5 minutes Max dose 3mg Adult ACLS Cardiac Arrest Amio dose: 1st: 300mg bolus 2nd: 150mg - ANSWER- Adult ACLS how many compressions? - ANSWER-At least 100 compressions a minute Vasopressin push dose & infusion dose - ANSWER-1 dose of 40 units 0.02-0.04 units per minute Thrombocytopenia - ANSWER-Platelet count of less than < 150,000 uL Trauma triad of death - ANSWER--Hypothermia: warm room, warm blanket, warmed fluid
ACLS VF or PULSELESS VT Amiodarone dose - ANSWER-Shock, CPR Epi 1 mg q 3-5 minutes Amio 300 mg bolus first dose Amio 150 mg Second dose calculating CCF code - ANSWER-Actual Chest compression time / Total code time ACLS Magnesium Drug facts - ANSWER-Na/K pump agonist Suppression on L- and T-type calcium channels and suppression of ventricular after- depolarizations ACLS Torsades with long QT interval providers should... - ANSWER-Magnesium Sulfate 1-2 g IV diluted in 10 mL Bolus over 20 minutes ACLS Septic Shock Tx - ANSWER-ABX! Don't Delay 30 mL/Kg crystalloid LR septic shock and Central VENOUS ScVO2 oxygen extraction LOW < 65% is bad. - ANSWER-Drawn from a CVC. Surrogate to Svo2. Normal oxygen extraction is 25 - 30% corresponding to a ScvO2 >65% < 65% = Impaired tissue oxygenation
80% = High PaO2; or suspect: — Cytotoxic dysoxia (e.g. cyanide poisoning, mitochrondial disease, severe sepsis) — Microcirculatory shunting (e.g. severe sepsis, liver failure, hyperthyroidism) — Left to right shunts Adult Dopamine IV Infusion Dose - ANSWER--Correct hypovolemia first
Arrest r/t Cardiac tamponade... - ANSWER-Give volume
38.3 (100.9F) or < 36 (96.8F) Heart Rate > 90 bpm
One Evidence of loss of cerebral auto regulation is - ANSWER-Cushings's response: Widening pulse pressure Reflex bradycardia Decreased respiratory efforts maintain a CPP of ______ to - ______ to optimize cerebral blood flow - ANSWER-Greater than 65 Usually 70- 90 Normal 60- 100 Acceptable 50-70 mm Hg How do you calculate MAP? - ANSWER-Systolic BP + (2 x DBP) / 3 Which leads look at the inferior surface of the heart? ie RV infarction inferior MI? - ANSWER-II III & aVF What does an inferior MI look like on EKG? - ANSWER- What are some physical exams you can perform to assess systemic vascular resistance? - ANSWER-pulse pressure variation What are Kinematics and why do we care? - ANSWER-Kinematics and the physics of energy transfer What is dynamic compliance? - ANSWER-Dynamic compliance is static compliance plus airway resistance. bronchospasm would affect dynamic compliance because it increases airway resistance.Differentiate between the factors that affect airway resistance and factors that affect expansion of the alveoli, lung, or chest wall. Dynamic compliance is affected by airway resistance factors. Commotio cordis is a phenomenon in which: - ANSWER-ventricular fibrillation is induced following blunt trauma to the chest during the heart's repolarization period. Ashman phenomenon - ANSWER-Aberrant conduction of a supraventricular beat commonly seen in patients with atrial fibrillation; wide SV beat after a QRS complex that is preceded by a long pause. Which of the following may be useful in systolic dysfunction but may be detrimental in diastolic dysfunction? A. β-blockers B. Angiotensin-converting enzyme inhibitors C. Aldosterone antagonists D.
Tricuspid valve D. Mitral valve In a pulmonary artery waveform the three components of the waveform are systole, dicrotic notch, and diastole. Systole is the pressure generated by the right ventricle so that the pulmonic valve will be pushed open, the dicrotic notch is caused by the closure of the pulmonic valve, and diastole is the pressure in the pulmonary artery during ventricular diastole. The diastolic pressure is a reflection of the vascular tone in the pulmonary vascular bed. If the vessels are constricted or if there is back pressure from the left side of the heart, the diastolic pressure will be high. A 36 yo patient presents in VTACH what is the most appropriate drug? A.Amiodarone (Cordarone) B.Verapamil (Calan) C.Adenosine (Adenocard) D.Ibutilide (Corvert) - ANSWER-The first drug for a wide QRS complex tachycardia is amiodarone. If the rhythm does not respond to amiodarone, synchronized cardioversion is indicated. Verapamil and adenosine typically are used for narrow QRS complex tachycardia. Eliminate options b and c. Ibutilide is used for acute-onset atrial fibrillation. This rhythm is regular, so eliminate option d and choose option a. An S4 is noted during cardiac auscultation. This sound indicates: - ANSWER-A. atrial contraction and propulsion of blood into a noncompliant ventricle. B. inflammation of the pericardium. C. opening of a defective semilunar valve. D. rapid ventricular filling into an already distended ventricle. An S4 occurs during the end of diastole when the atria contract but the ventricle is noncompliant. An S4 occurs in myocardial ischemia, infarction, and hypertrophy. Most patients with an acute myocardial infarction have an S4 for the first 48 hours. What are signs of hypoperfusion after a rhythym change? - ANSWER-Assess the patient for clinical indications of hypoperfusion (e.g., cool skin, decreased urine output, narrowed pulse pressure, and hypotension). A lateral Wall MI will be seen as St elevations in which leads, and stems from a blockage in which coronary arteries? - ANSWER-1. High Lateral: lead 1 and AVL V5 and V6 are low lateral wall... ischemia from the circumflex artery.
In Acute coronary Syndromes unstable angina is characterized by...... - ANSWER-- Ischemic Symptoms suggestive of ACS